Max in WT synaptoneurosomes, suggesting that Src signaling could possibly be downregulated in KI synapses. Conversely, our ability to rescue SERT functionality in KI midbrain synaptoneurosomes by the inhibition of FAK indicates elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by improved pFAK localization in five-HT synapses. Our https://shermanc813rwq9.wikisona.com/user